New Data at ERA-EDTA Show Mimpara® (Cinacalcet) Improves Management of Secondary Hyperparathyroidism in Daily Clinical Practice



    Amgen (Europe) GmbH today announced results from the Evaluation

    of the Clinical Use of Mimpara in Haemodialysis

    and Peritoneal Dialysis Patients, an Observational

    Study (ECHO) at the European Renal Association "“ European Dialysis and Transplant Association (ERA-EDTA) congress. Data

    show that a higher percentage of dialysis patients with secondary

    hyperparathyroidism (SHPT) achieved and maintained National

    Kidney Foundation Kidney Disease Outcomes Quality Initiative (KDOQITM)

    targets with Mimpara® (cinacalcet), compared to their previous treatment (vitamin D sterols

    and phosphate binders). In addition, data also show that Mimpara

    effectively controls SHPT regardless of whether patients were receiving

    concomitant vitamin D.
    "The achievement and maintenance of KDOQI targets is desirable for

    achieving optimal SHPT patient management. For the first time, these new

    final data demonstrate cinacalcet´s effectiveness in daily clinical

    practice, confirming that improvements seen with cinacalcet in

    randomised clinical trials can be reproduced in the clinic," said Doctor

    Neil Ashman, consultant nephrologist and honorary senior lecturer, Renal

    Unit, Barts and the London NHS Trust, London, UK.
    Results from this pan-European observational study demonstrate that a

    higher percentage of SHPT patients (n=1865) achieved and maintained

    KDOQI targets for all four parameters - parathyroid hormone (iPTH)

    phosphorus (P), calcium (Ca) and calcium phosphorus product (Ca x P) -

    for up to 12 months following initiation of Mimpara, compared to their

    previous treatment (standard care: vitamin D sterols and phosphate

    binders). KDOQI targets include iPTH 150-300 pg/ml, 3.5-5.5 mg/dl, Ca

    8.4-9.5 mg/dl, Ca x P <55 mg2/dl2.
    Mimpara improved attainment of targets in a patient population with

    severe SHPT (median iPTH was 721 pg/mL and 54% of patients had

    uncontrolled Ca x P at baseline). In addition, only a small proportion

    of patients on traditional therapy were able to sustain KDOQI targets in

    the six months prior to the introduction of cinacalcet despite the

    extensive use of vitamin D and phosphate binders. These results are

    important because the role of vitamin D in controlling SHPT in dialysis

    patients may be limited by the development of hypercalcemia and

    hyperphosphatemia.
    Adverse events related to treatment were reported in 11.3% of patients.

    The most common adverse events were nausea (4.6%) and vomiting (3.1%).

    Six patients (0.3%) experienced serious adverse events and there were no

    treatment-related deaths. 75.5% of patients remained on Mimpara at end

    of study; the main reason for discontinuation was renal transplantation

    (5.2% of patients).
    About the ECHO Study
    ECHO is a pan-European, multicenter, observational study that explored

    Mimpara use in daily clinical practice. The study enrolled 1865 SHPT and

    dialysis patients between July 2005 and October 2006 from 187 sites in

    12 countries. Patients on dialysis were enrolled six months before and

    up to 12 months after initiating treatment with cinacalcet.
    About SHPT
    Secondary hyperparathyroidism (SHPT) is a metabolic disorder that

    develops in chronic kidney disease (CKD) patients on dialysis and

    results in increased secretion of parathyroid hormone (PTH), which may

    lead to bone disease, bone pain and fractures, cardiovascular and soft

    tissue calcification and parathyroid hyperplasia.
    Patients develop SHPT as their kidneys fail because their ability to

    excrete phosphorus and produce active vitamin D diminishes, leading to a

    fall in blood calcium levels. Additionally, a low level of calcium

    results in stimulating the parathyroid gland to secrete more PTH in an

    attempt to normalise blood levels of calcium. Over time, continuous PTH

    secretion leads to excessive growth of the parathyroid gland, high

    levels of PTH, calcium and phosphorus, and HPT complications including

    bone disease and soft tissue and vascular calcification, which increases

    the risk for cardiovascular events.
    The majority of an estimated 324,000 CKD patients on dialysis in Europe

    suffers from some degree of SHPT. According to the Dialysis Outcomes and

    Practice Patterns Study (DOPPS) 26 percent of CKD patients on dialysis

    throughout the world had PTH levels above the KDOQI guidelines (<33.0

    pmol/L or 300 pg/ml), an indication of SHPT.
    About Mimpara
    Mimpara (also known as Sensipar® in the

    United States, Australia, New Zealand and Canada) is a calcimimetic

    agent that is approved for the treatment of secondary

    hyperparathyroidism (SHPT) in patients with chronic kidney disease

    receiving dialysis.
    Calcimimetics amplify the action of calcium on the calcium-sensing

    receptors on the parathyroid gland, thereby decreasing the secretion of

    PTH. Mimpara is the first and only calcimimetic agent approved for use

    in dialysis patients to specifically bind to and directly modulate the

    calcium-sensing receptor on the surface of the chief cell of the

    parathyroid gland while simultaneously lowering blood calcium and

    phosphorus levels. In dialysis patients, Mimpara significantly reduces

    PTH levels while simultaneously lowering blood calcium and phosphorus

    levels. Mimpara binds to the parathyroid gland´s

    calcium-sensing receptors, making them more sensitive to calcium, which

    causes the gland to reduce its release of PTH.
    The threshold for seizures may be lowered by reductions in calcium

    levels and, infrequently, seizures have been reported with use of

    Mimpara. The most commonly reported side effects are nausea and

    vomiting. In post-marketing safety surveillance, isolated, idiosyncratic

    cases of hypotension and/or worsening heart failure have been reported

    in patients with impaired cardiac function, in which a causal

    relationship to Mimpara could not be completely excluded and may be

    mediated by reductions in serum calcium levels.
    About Amgen
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